#YIP MAN 2 OVERDOSE FULL#

The patient denied flu symptoms in the previous days (This excludes infectious causes of acute muscle weakness as Guillan-Barre syndrome) or a family history of muscle dystrophies or neurologic diseases (This excludes familial muscle dystrophies as a cause of hypotonia).Carboxyhaemoglobin level was investigated and found to be 1.8% (Normal range is 1-2 % in non-smokers). Neurologic examination revealed hypotonia and impaired reflexes in both lower limbs, sparing the upper limbs.

Arterial blood gas (ABG) results showed PH 7.36, PCO 2 35 mmHg, PO 2 99 mmHg, HCO 324 mEq/L. ECG revealed normal sinus rhythm without ischaemic changes. Finger stick bedside blood glucose was 86 mg/dl. Chest examination revealed bilateral vesicular breath sounds and clear chest. Pupils were rounded, regular, reactive, equal and central.

#YIP MAN 2 OVERDOSE FULL#

regular, full RR 20 breath/min., rectal temperature 37вЊ. On examination, the patient was fully conscious with stable vital signs. The patient was referred to the Toxicology Unit at Mansoura University Emergency Hospital. The provisional diagnosis made by the referring physician was вЂFood PoisoningвЂ™. The patient received fluids therapy and O 2 inhalation and felt better while his parents were in coma and suffering from respiratory depression and are still hospitalized in ICU on mechanical ventilation, as given by history. The condition started since one day after sharing a meal of fish and then his parents were found at home by the neighbours in a bad condition and were transferred to the nearest hospital. Timely prompt medical care involving team therapy is necessary to prevent further complications like RF and muscle wasting.ДЄ 34-years old non-smoker Egyptian male who is married and has two offspring and is working in mechanics repair, came to the Emergency Department of Mansoura University Hospital on Tuesday 15 th January 2013 seeking the medical care for his inability to walk or stand steadily. Conclusion : Carbon monoxide poisoning should be suspected in patients presented with acute illness without prior medical or surgical cause, and rare complications of CO poisoning like rhabdomyolysis should be suspected particularly in patients with delayed seek of medical care. The patient underwent rehabilitation therapy to improve his neuromuscular state. After one month, levels of serum creatinine and alanine transaminase became normal. The patient begins haemodialysis with follow up of his serum K +, creatinine and CK. Based on the above-mentioned data, a diagnosis of rhabdomyolysis complicating acute CO exposure with secondary renal insult was made. A work-up that involved appropriate imaging and serum creatinekinase (CK) measurement revealed extremely elevated serum CK, normal appearance of liver and increased echogenicity of both kidneys with preserved cortico-medullary differentiation. Investigations revealed increased serum alanine transaminase and serum creatinine. No history of toxin exposure but a condition of sudden acute illness affecting him and his parents and upon which his old-age parents, were transferred to ICUin coma. Neurologic examination revealed hypotonia and diminished reflexes in both lower limbs. However, the patient cannot stand steadily or walk. Initial examination revealed a fully conscious patient with stable vital signs and arterial blood gases. Case presentation : A 34-years old Egyptian male was referred to the Toxicology Unit from the ED for evaluation of his lower limb weakness.

Rhabdomyolysis, compartment syndrome, renal failure and peripheral neuropathy are unusual complications of CO That can be faced during practice and should be considered by physicians. Moderate to severe CO poisoning produces significant morbidity and mortality that provokes treatment controversy. Mild poisoning by CO can be Mistaken for a non-specific headache or a viral illness headache or viral illness. Introduction : Carbon Monoxide (CO) is a colourless, odourless, tasteless gas.

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